Original articleBerger disease: Henoch-Schönlein syndrome without the rash
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Immunoglobulin A Nephropathy and Immunoglobulin A Vasculitis
2019, Pediatric Clinics of North AmericaGenetic Determinants of IgA Nephropathy: Western Perspective
2018, Seminars in NephrologyCitation Excerpt :Pedigree analyses also have suggested an etiologic connection between IgAN and other potentially related diseases by showing co-segregation. For example, there are reports of families in which some relatives are affected by IgAN while others have Henoch-Schönlein purpura (HSP),26 leading to the hypothesis that IgAN is a subtype of HSP without the vascular involvement.27-30 In addition, family-based observations provided support for an immune component of IgAN by showing co-clustering of microhematuria, a symptom of IgAN, and serum immunologic abnormalities,31 including increased cytokine levels.32
Recent progress in Berger's disease (immunoglobulin A nephropathy)
2017, Bulletin de l'Academie Nationale de MedecineUrinary myeloid IgA Fc alpha receptor (CD89) and transglutaminase-2 as new biomarkers for active IgA nephropathy and henoch-Schönlein purpura nephritis
2016, BBA ClinicalCitation Excerpt :Actually, up to 30% of IgAN patients progress to ESRD by 20 years, and many of these patients need for renal replacement treatment [6,7]. The clinical, genetic and immunologic characteristics of IgAN and HSPN are closely linked, and HSPN can be considered as the systemic form of IgAN [4,8,9]. IgAN and HSPN share a common and central pathogenic mechanism associated with an aberrant O-linked glycosylation in the hinge region of IgA1 molecules [2,4,5,10,11].
IgA Nephropathy and Related Diseases
2015, Mucosal Immunology: Fourth EditionA narrative review of potential drug treatments for nephritis in children with IgA vasculitis (HSP)
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