Elsevier

American Heart Journal

Volume 109, Issue 1, January 1985, Pages 164-167
American Heart Journal

Brief communication
Torsade de pointes and magnesium deficiency

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    Magnesium deficiency impairs sodium-potassium adenosine triphosphatase, leading to a decrease in intracellular potassium and a relatively depolarized resting membrane potential, predisposing to arrhythmia. In nondialysis patients, there are anecdotal reports of monomorphic ventricular tachycardia, torsades de pointes, and ventricular fibrillation associated with hypomagnesemia that responded to magnesium repletion.46,47 Concomitant potassium depletion further increases the risk, and use of a low dialysate potassium concentration may further increase the risk of low dialysate magnesium concentration, though this has not been studied.

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The opinions or assertions contained herein are the private views of the authors and are not to be construed as official or as reflecting the views of the Department of the Army or the Department of Defense.

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