Regular ArticleCell Death, Axonal Damage, and Cell Birth in the Immature Rat Brain Following Induction of Hydrocephalus
References (62)
- et al.
Central neuron-glial and glial–glial interactions following axon injury
Prog. Neurobiol.
(1998) A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein dye binding
Anal. Biochem.
(1976)- et al.
Nestin mRNA expression correlates with the central nervous system progenitor cell state in many, but not all, regions of developing central nervous system
Dev. Brain Res.
(1995) - et al.
Magnetic resonance imaging and behavioral analysis of immature rats with kaolin-induced hydrocephalus: pre- and postshunting observations
Exp. Neurol.
(1997) - et al.
Monoamine neurotransmitters and amino acids in the cerebrum and striatum of immature rats with kaolin-induced hydrocephalus
Brain Res.
(1998) - et al.
Endogenous progenitors remyelinate demyelinated axons in the adult CNS
Neuron
(1997) Distribution of commissural axon terminals in the rat neocortex
Exp. Neurol.
(1970)- et al.
Neonatal hydrocephalus. Mechanisms and consequences
Neurosurg. Clin. North Am.
(1998) - et al.
Use of bromodeoxyuridine-immunohistochemistry to examine the proliferation, migration and time of origin of cells in the central nervous system
Brain Res.
(1988) - et al.
Neural stem cells in the adult mammalian forebrain: A relatively quiescent subpopulation of subependymal cells
Neuron
(1994)
The origin and nature of beading: A reversible transformation of the shape of nerve fibers
Prog. Neurobiol.
(1997)
Increased proliferation of precursor cells in the adult rat brain after targeted lesioning
Brain Res.
(1996)
The effects of hydrocephalus upon the developing brain. Histological and quantitative studies of the ependyma and subependyma in hydrocephalic rats
J. Neurol. Sci.
(1978)
Growth associated protein-43 is increased in cerebrum of immature rats following induction of hydrocephalus
Neuroscience
(1998)
Cellular and molecular pathogenesis of periventricular white matter injury
Ment. Retard. Dev. Disabil. Res. Rev.
(1997)
Protease antigen recovery decreases the specificity of bromodeoxyuridine detection in formalin-fixed tissue
J. Histochem. Cytochem.
(1997)
Multiple extracellular signals are required for long-term oligodendrocyte survival
Development
(1993)
Ferritin, transferrin, and iron in selected regions of the adult and aged rat brain
J. Comp. Neurol.
(1993)
Mitotic activity in the brain of the adult rat
Anat. Rec.
(1959)
A cautionary note on the use of the TUNEL stain to determine apoptosis
NeuroReport
(1995)
A rapid method to determine proliferation patterns of normal and malignant tissues by H3 mRNAin situ
Am. J. Pathol.
(1990)
Anaerobic glycolysis preceding white-matter destruction in experimental neonatal hydrocephalus
J. Neurosurg.
(1994)
Reactive astrocytes express the embryonic intermediate filament nestin
Neuroreport
(1994)
The chronology of oligodendrocyte differentiation in the rat optic nerve: Evidence for a signaling step initiating myelination in the CNS
J. Neurosci.
(1995)
Subependymal cells provide a faster response to ependymal injury than astrocytes in the hydrocephalic brain
Neuropathol. Appl. Neurobiol.
(1992)
In vivo
J. Neurosci.
(1996)
Neuropathological changes caused by hydrocephalus
Acta Neuropathol.
(1993)
Periventricular pathology in hydrocephalic rabbits before and after shunting
Acta Neuropathol.
(1988)
Neuropathological changes in chronic adult hydrocephalus: Cortical biopsies and autopsy findings
Can. J. Neurol. Sci.
(1997)
Acute and chronic cerebral white matter damage in neonatal hydrocephalus
Can. J. Neurol. Sci.
(1994)
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Comprehensive analysis of differentially expressed profiles of long non-coding RNAs and messenger RNAs in kaolin-induced hydrocephalus
2019, GeneCitation Excerpt :Our functional enrichment analysis suggested that complement activation may be critical in the pathophysiological process of hydrocephalus, and the dysregulated lncRNAs associated with complement activation may play a role in epigenetic regulation. Astrocytes are activated during hydrocephalic brain injury, and GFAP (an intermediate filament and a key component of the astrocyte cytoskeleton) increases in white matter, especially in periventricular white matter (Del Bigio and Zhang, 1998; Lopes Lda et al., 2009; Deren et al., 2010; Olopade et al., 2012). However, whether astroglial activation is better or worse for brain injuries is controversial.
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